Small Animal Medical Differential Diagnosis: A Book of by Mark Thompson DVM DABVP(Canine and Feline)

By Mark Thompson DVM DABVP(Canine and Feline)

Small Animal scientific Differential prognosis, second Edition is a realistic, concise consultant to the differential prognosis, etiology, laboratory abnormalities, and class of scientific indicators and scientific problems in canines and cats. by way of protecting approximately each attainable signal and medical sickness correct to small animal scientific perform, this pocket-sized, fast reference is helping you're making extra trustworthy on-the-scene decisions.

  • More than four hundred lists bring crucial clinical diagnostic information from a number of assets into a single rapid reference.
  • An equipped presentation of differential diagnoses by means of signal and symptom, sickness, and physique method, facilitates quickly and versatile entry to details at many levels of the diagnostic work-up.
  • Alphabetical directory of all correct laboratory exams makes details effortless to discover for college kids and skilled practitioners alike.
  • Easily establish the likeliest prognosis by reviewing the probabilities indexed so as of incidence.
  • Pocket-sized for portability, practicality, and fast reference.

  • NEW! insurance of latest issues and syndromes expands the span of differential diagnoses that can assist you successfully review extra indicators and symptoms.
  • NEW! Addition of recent and extra generic diagnostic and laboratory exams keeps you recent as lab exams turn into extra really expert and sophisticated.
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Additional resources for Small Animal Medical Differential Diagnosis: A Book of Lists, 2e

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Pigmented tumors—apocrine cysts are bluish, cutaneous hemangiomas and hemangiosarcomas appear red, dark purple, or bluish-black. Melanomas, melanocytomas, and basal cell tumors are frequently black. Squamous cell carcinomas, trichoblastomas, and fibromas also may be dark brown to black. Hyperthermia Fever Exogenous pyrogens (infectious agents and their products, inflammation or necrosis of tissue, immune complexes, pharmacologic agents, bile acids) Endogenous pyrogens (fever-producing cytokines) Heat Stroke High ambient temperatures Exercise Poor ventilation Brachycephalic conformation Obesity Exercise Hyperthermia Sustained exercise Seizure disorders (especially prolonged or cluster seizures) Hypocalcemic tetany (eclampsia) Pathologic Etiologies Lesions in or around anterior hypothalamus Hypermetabolic disorders PART ONE Clinical Signs Approach to Differential Diagnosis 41 Hyperthyroidism Pheochromocytoma Malignant hyperthermia Halothane Succinylcholine Hypopigmentation Due to melanocyte destruction, dysfunction, or abnormal distribution of melanosomes.

Seen most commonly in Siamese cat, Belgian Tervuren, German Shepherd, collie, Rottweiler, Doberman Pinscher, Giant Schnauzer. Nasal hypopigmentation—season-associated lightening of nasal planum during winter months most common in Siberian Husky, Golden Retriever, Labrador Retriever, and Bernese Mountain Dog. Seen also in many other breeds. Acquired Hypopigmentation Postinflammatory—Discoid lupus erythematosus is the most common cause of postinflammatory nasal depigmentation. Also pemphigus complex, SLE, uveodermatologic syndrome, bullous pemphigoid, mucocutaneous pyoderma, drug eruption, and contact dermatitis.

Fragmentation (disseminated intravascular coagulation, heartworm disease, hemangiosarcoma, vasculitis, hemolytic uremic syndrome, diabetes mellitus) Pyruvate kinase deficiency Phosphofructokinase deficiency Feline porphyria Copper toxicity Neonatal isoerythrolysis Oxidative injury (onions, acetaminophen, zinc, benzocaine, mothballs, phenazopyridine) Blood loss (external blood loss, blood loss to a body cavity, coagulopathies, endoparasites, gastrointestinal blood loss) Nonregenerative Anemia Anemia of chronic disease Anemia from renal failure 56 PART ONE Clinical Signs Approach to Differential Diagnosis Feline leukemia virus (FeLV) Endocrine (mild anemia associated with hypoadrenocorticism, hypothyroidism) Myeloaplasia/aplastic anemia (FeLV infection, ehrlichiosis, trimethoprim-sulfa, estrogen toxicity, phenylbutazone, chemotherapy, chloramphenicol) Myelodysplasia Myeloproliferative and lymphoproliferative disorders Myelofibrosis Shock Cardiogenic Decreased ventricular function • Dilated cardiomyopathy • Myocarditis • Myocardial infarction Compromised ventricular filling • Hypertrophic cardiomyopathy • Cardiac tamponade Severe endocardiosis Outflow obstruction • Intracardiac tumors • Aortic stenosis • Hypertrophic obstructive cardiomyopathy • Heartworm disease • Thrombosis • Severe arrhythmia Noncardiogenic Trauma Hypovolemia • Severe blood loss • Dehydration • Hypoadrenocorticism Disruptions in blood flow • Sepsis and endotoxemia • Hypotension Papules and Pustules • • • • • • Bacterial pyoderma (papules and pustules) Demodicosis (papules and pustules) Dermatophytosis (rare papules, uncommon pustules) Sarcoptes mange (papules, no pustules) Cheyletiellosis (rare papules, no pustules) Otacariosis (rare papules, no pustules) PART ONE • • • • Clinical Signs Approach to Differential Diagnosis Trombiculosis (papules, rare pustules) Hypersensitivity (papules, rare pustules) Pemphigus (papules and pustules) Early-stage neoplasia (papules, no pustules) Paresis and Paralysis Upper Motor Neuron Tetraparesis or hemiparesis • Severe forebrain lesion • Brain stem lesion • First to fifth cervical (C1-C5) spinal lesion Paraparesis or rear limb monoparesis • Third thoracic to third lumbar (T3-L3) spinal lesion Lower Motor Neuron Tetraparesis Generalized lower motor neuron disease • Flaccid paresis/paralysis • Acute polyradiculoneuritis/“coonhound paralysis” • Tick paralysis • Botulism • Myasthenia gravis • Toxicants • Coral snake • Black widow spider • Herbicides (2,4 D) • Macadamia nuts Paraparesis • Fourth lumbar to second sacral (L4-S2) spinal lesion Hemiparesis with lower motor neuron forelimb • Sixth cervical to second thoracic (C6-T2) spinal lesion Aortic thromboembolism Degenerative myelopathy Monoparesis Peripheral nerve lesion Petechiae and Ecchymoses Thrombocytopenia Increased Platelet Destruction Immune-mediated thrombocytopenia Systemic lupus erythematosus (SLE) Heartworm disease 57 58 PART ONE Clinical Signs Approach to Differential Diagnosis Decreased Platelet Production Bone Marrow Suppression Infectious disease (ehrlichiosis, babesiosis, Rocky Mountain spotted fever, leishmaniasis, feline leukemia virus, feline immunodeficiency virus) Neoplasia Drug reactions Myeloproliferative disease Virus-associated myelodysplasia Estrogen toxicity Consumption of Platelets Disseminated intravascular coagulation (DIC) Vasculitis Sequestration of Platelets (Unlikely to Cause Clinical Signs) Splenomegaly Hepatomegaly Endotoxemia Thrombopathia Inherited Cocker Spaniel, Otterhound, Great Pyrenees, Bassett Hound, American Cocker Spaniel, cats Acquired Drugs (aspirin, cephalothin, carprofen, hydroxyethyl starch) Uremia Liver disease Dysproteinemias Von Willebrand Disease Lack of von Willebrand factor leads to impaired platelet adhesion.

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