By Beullens A.
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Later studies were concerned with mapping symptom determinants, and an inability to infect a host systemically was considered one type of symptom determinant. There is a subtle difference in designating a viral gene product as a symptom determinant rather than an Avr gene; consequently these studies generally were not included in early reviews that discuss the characterization of Avr genes (Keen and Staskawicz, 1988). However, they clearly laid the A2. Viral determinants of resistance versus susceptibility 17 TABLE 1: Identification of virus genetic components that act as host range determinants: studies conducted before the availability of infectious virus clones.
Application for rhizobacteria in transplant production and yield enhancement. Acta Hort. 631:219-229. Loebenstein, G. and Lovrekovich, L. 1966. Interference with tobacco mosaic virus local lesion formation in tobacco by injecting heat-killed cells of Pseudomonas syringae. Virology 30:587-591. W. 1969. Inhibition of tobacco mosaic virus by a bacterial extract. Phytopathology 59:658-662. , Metraux, J. , and Defago, G. 1994. Induction of systemic resistance of tobacco to tobacco necrosis virus by the root-colonizing Pseudomonas fluorescens strain CHA0: Influence of the gacA gene and of pyoverdine production.
Mixtures of plant growth-promoting rhizobacteria enhance biological control of multiple cucumber pathogens. Phytopathology 88:1158-1164. W. 1996. Induced systemic resistance against cucumber mosaic cucumovirus using plant growthpromoting rhizobacteria (PGPR). Plant Dis. 80:891-894. F. 1961. Systemic acquired resistance induced by localized virus infections in plants. Virology 14:340-358. , and Lester, D. 1998. A multivirus epidemic of tomatoes in Alabama. Plant Dis. 82:117-120. , Gilliland, A. P.