By Kazumasa Aoyagi, Yutaka Kuzure (auth.), Joseph F. Clark (eds.)
Guanidino compounds include Creatine, Arginine, and the Guanidines. some time past years there were over 2000 released articles with the names of those compounds within the name. it is easy to visit any foodstuff or future health meals shop and purchase those as vitamins since it is assumed they increase healthiness and athletic performance.
This quantity comprises an up-to-the-minute precis of the medical and scientific facets of basically all of the biologically energetic Guanidino Compounds. The articles summarize the present clinical wisdom of those compounds on the subject of proper medical stipulations, and talk about the chemical, organic, and medical services of those compounds.
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Additional info for Guanidino Compounds in Biology and Medicine
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Am J Physiol248: C320C329, 1985 26. Nakayama S, Seo Y, Takai A, Tomita T, Watari H: Phosphorous compounds studied by 31p nuclear magnetic resonance spectroscopy in the taenia of guinea-pig caecum. J Physiol 402: 565-578, 1988 27. Ashoori F, Takai A, Tokuno H, Tomita T: Effects of glucose removal and rcadmission on the potassium contracture in the guinea-pig taenia coli. J Physiol 356: 33--48, 1984 28. Katayama N, Huang S-M, Tomita T, Brading AF: Effects of cromakalim on the electrical slow wave in the circular muscle of guinea-pig gastric antrum.
Marescau, LA. Qureshi, A. Mori (eds). Guanidino Compounds in Biology and Medicine 2. John Libbey, London, 1997, pp 273-277 29. Aoyagi K, Akiyama K, Shahrzad S, Tomida C, Hirayama A, Nagase S, Takemura K, Koyama A, Ohba S, Narita M: Formation of guanidinosuccinic acid, a stahle nitric oxide mimic, from argininosuccinic acid and nitric oxide-derived free radicals. Free Radic Res 31: 59--65, 1999 30. Aoyagi K: Inhibition of arginine synthesis by urea: a ncw mechanism of arginine deficiency in renal failure which leads to increased hydroxyl radical generation.
Fig. 3. Argininosuccinic acid or ASA. Point B is the normal cleavage point lyase, known as argininosuccinic aciduria, GSA is not found in the urine. It is similarly absent in all other reported cases of inborn errors of the urea cycle [5, 24, 25] (Fig. 5). Can oxygen, or more specifically reactive oxygen species, represent the rate limiting factor in the GSA shunt? It has been proposed that uremia is a particularly oxidative state because of the loss of renal clearance of superoxide anions [23, 26].