By Edward C. Feldman DVM DACVIM, Richard W. Nelson DVM, Visit Amazon's Claudia Reusch Page, search results, Learn about Author Central, Claudia Reusch, , J. Catharine Scott-Moncrieff
No different identify deals such commitment to the intensity, adventure, and concentration of endocrinology as Canine and pussycat Endocrinology, 4th Edition. entire assurance comprises almost each universal and unusual situation in endocrinology, plus the main up-to-date info on foodstuff, geriatric care, pathophysiology, checking out approaches, and cost effective and expedient diagnostic protocols. With its logical, step by step counsel for choice making, analysis, and prescribing, you may be well-equipped to deal with the broad spectrum of endocrine and metabolic issues in canines and cats.
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Additional info for Canine and Feline Endocrinology, 4e
0-mL bottles containing 100 μg DDAVP/mL) is used most commonly for treating CDI in humans and is effective for the treatment of CDI in dogs and cats (Harb et al, 1996). Administration of DDAVP nasal to dogs and cats via the intranasal route is possible but not recommended. Increase contact with humans or dogs CDI, Central diabetes insipidus; DDAVP, desmopressin acetate; LVP, lysine vasopressin; NDI, nephrogenic diabetes insipidus; SC, subcutaneous. transferred to a sterile eye dropper bottle and drops placed into the conjunctival sac of the dog or cat.
In humans, SIADH has been observed in a variety of disorders, particularly pulmonary, CNS, and neoplastic disorders (Box 1-7) (Robinson and Verbalis, 2011). The most common association of SIADH is with tumors, most notably bronchogenic carcinomas. Vasopressin or a peptide having comparable biologic activity is produced by tumors. SIADH is rare in the dog and cat and has been reported in one dog with heartworm disease, one dog with liver disease, one dog with an undifferentiated carcinoma, one dog with a tumor in the region of the hypothalamus, one cat following anesthesia, laparoscopy and metoclopramide treatment, and was considered idiopathic in two dogs (Rijnberk et al, 1988; Houston et al, 1989; Cameron and Gallagher, 2010; Kang and Park, 2012).
The diagnosis was initially based on results of a modiﬁed water deprivation test similar to that illustrated in Fig. 1-22. However, sophisticated studies have revealed that some individuals have a metabolic explanation for such a test result. , an abnormally elevated set point in their osmoreceptors for stimulating release of AVP). Therefore, at a relatively high plasma osmolality, which should cause release of AVP, these individuals remain “AVP free,” polyuric, and thus polydipsic. , thirst may be stimulated at a plasma osmolality of 290 pOsm when it should not be stimulated until the osmolality reaches 295 or 300 pOsm).